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Intraclonal variations among Streptococcus pneumoniae isolates influence the likelihood of invasive disease in children.

Journal article
Authors Sarah Browall
Martin Norman
Jeanette Tångrot
Ilias Galanis
Karin Sjöström
Jessica Dagerhamn
Christel Hellberg
Anuj Pathak
Tiziana Spadafina
Andreas Sandgren
Patrick Bättig
Oscar Franzén
Björn Andersson
Åke Örtqvist
Staffan Normark
Birgitta Henriques-Normark
Published in The Journal of infectious diseases
Volume 209
Issue 3
Pages 377-88
ISSN 1537-6613
Publication year 2014
Published at
Pages 377-88
Language en
Keywords Adolescent, Animals, Antigens, Bacterial, analysis, Carrier State, epidemiology, microbiology, Child, Child, Preschool, Disease Models, Animal, Electrophoresis, Gel, Pulsed-Field, Female, Genetic Variation, Genome, Bacterial, Genotype, Humans, Infant, Male, Membrane Proteins, analysis, Mice, Mice, Inbred C57BL, Molecular Typing, Pneumococcal Infections, epidemiology, microbiology, pathology, Prophages, genetics, Sequence Analysis, DNA, Streptococcus Phages, genetics, Streptococcus pneumoniae, classification, genetics, isolation & purification, Virulence
Subject categories Infectious Medicine, Pediatrics

Abstract

Pneumococcal serotypes are represented by a varying number of clonal lineages with different genetic contents, potentially affecting invasiveness. However, genetic variation within the same genetic lineage may be larger than anticipated.

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